Table of Contents Table of Contents
Previous Page  100 / 2894 Next Page
Information
Show Menu
Previous Page 100 / 2894 Next Page
Page Background

79. From RP, Warner DS, Todd MM, et al: Anesthesia

for craniotomy: A double blind comparison of

alfentanil, fentanyl and sufentanil. Anesthesiology

73:896-904, 1990.

80. Keykhah MM, Smith DS, Carlsson C, et al:

Influence of sufentanil on cerebral metabolism and

circulation in the rat. Anesthesiology 63:274-277,

1985.

81. Werner C, Hoffman WE, Baughman VL, et al:

Effects of sufentanil on cerebral blood flow, cerebral

blood flow velocity and metabolism in dogs.Anesth

Analg 72:177-181, 1991.

82. Murkin JM, Farrar JK, TweedWA: Sufentanil anaes-

thesia reduces cerebral blood flow and cerebral

oxygen consumption [abstract]. Can J Anaesth

35:S131, 1988.

83. Mayer N, Weinstabl C, Podreka I, et al: Sufentanil

does not increase cerebral blood flow in healthy

human volunteers. Anesthesiology 73:240-243,

1990.

84. Weinstabl C, Mayer N, Spiss CK: Sufentanil decrea-

ses cerebral blood flow velocity in patients with

elevated intracranial pressure. Eur J Anaesthesiol

9:481-484, 1992.

85. Weinstabl C, Mayer N, Richling B, et al: Effect of

sufentanil on intracranial pressure in neurosurgical

patients. Anaesthesia 46:837-840, 1991.

86. Werner C, Kochs E, Hoffman WE, et al: Sufentanil

does not change cerebral hemodynamics and ICP

in head injured patients [abstract]. J Neurosurg

Anaesthesiol 4:313, 1992.

87. Jamali S, Ravussin P, Archer D, et al: The effects of

bolus administration of opioids on cerebrospinal

fluid pressure in patients with supratentorial lesions.

Anesth Analg 82:600-606, 1996.

88. Lauer KK, Connolly LA, Schmeling WT: Opioid

sedation does not alter intracranial pressure in

head injured patients. Can J Anaesth 44:929-933,

1997.

89. Sperry RJ, Bailey PL, Reichman MV, et al: Fentanyl

and sufentanil increase intracranial pressure in

head trauma patients. Anesthesiology 77:416-420,

1992.

90. Werner C, Kochs E, Bause H, et al: Effects of sufen-

tanil on cerebral hemodynamics and intracranial

pressure in patients with brain injury. Anesthesio-

logy 83:721-726, 1995.

91. Bristow A, Shalev D, Rice B, et al: Low-dose synthe-

tic narcotic infusions for cerebral relaxation during

craniotomies. Anesth Analg 66:413-416, 1987.

92. Shupak RC, Harp JR: Comparison between high-

dose sufentanil-oxygen and high-dose fentanyl-

oxygen for neuroanaesthesia. Br J Anaesth 57:375-

381, 1985.

93. Warner DS, Hindman BJ, Todd MM, et al: Intra-

cranial pressure and hemodynamic effects of remi-

fentanil versus alfentanil in patients undergoing

supratentorial craniotomy. Anesth Analg 83:348-

353, 1996.

94. Ostapkovich N, Baker KZ, Fogerty-Mack P, et al:

Cerebral blood flow and CO

2

reactivity is similar

during remifentanil/N

2

O and fentanyl/N

2

O anes-

thesia. Anesthesiology 89:358-363, 1998.

95. Wagner K,Wiloch F,Kochs E,et al: Dose-dependent

regional cerebral blood flow changes during remi-

fentanil infusion in humans. Anesthesiology 94:

732-739, 2001.

96. Lorenz I, Kolbitsch C, Hormann C, et al: The

influence of nitrous oxide and remifentanil on cere-

bral hemodynamics in conscious human volunteers.

Neuroimage 17:1056-1064, 2002.

97. Kofke WA, Blissitt PA, Rao H, et al: Remifentanil-

induced cerebral blood flow effects in normal

humans: Dose and ApoE genotype. Anesth Analg

105:167-175, 2007.

98. Ostapkovich ND, Baker KZ, Fogarty-Mack P, et al:

Cerebral blood flow and CO

2

reactivity is similar

during remifentanil/N

2

O and fentanyl/N

2

O anes-

thesia. Anesthesiology 89:358-363, 1998.

99. Forster A, Juge O, Louis M, et al: Effects of a specific

benzodiazepine antagonist (RO 15-1788) on cere-

bral blood flow. Anesth Analg 66:309-313, 1987.

100. Veselis RA, Reinsel RA, Beattie BJ, et al: Midazolam

changes cerebral blood flow in discrete brain

regions: an H

2

15

O positron emission tomography

study. Anesthesiology 87:1106-1117, 1997.

101. Forster A, Juge O, Morel D: Effects of midazolam on

cerebral hemodynamics and cerebral vasomotor

responsiveness to carbon dioxide. J Cereb Blood

Flow Metab 3:246-249, 1983.

102. Wolf J, Friberg L, Jensen J, et al: The effect of the

benzodiazepine antagonist flumazenil on regional

cerebral blood flow in human volunteers. Br J

Anaesth 34:628-631, 1990.

103. Knudsen L, Cold GE, Holdgard HO, et al: Effects of

flumazenil on cerebral blood flow and oxygen con-

sumption after midazolam anaesthesia for cranio-

tomy. Br J Anaesth 67:277-280, 1991.

104. Chiolero RL, Ravussin P, Anderes JP, et al: The

effects of midazolam reversal by RO 15-1788 on

cerebral perfusion pressure in patients with severe

head injury. Intensive Care Med 14:196-200, 1988.

105. Misfeldt BB, Jorgensen PB, Spotoft H, et al: The

effects of droperidol and fentanyl on intracranial

pressure and cerebral perfusion pressure in neu-

rosurgical patients. Br J Anaesth 48:963-968,

1976.

106. Michenfelder JD, Theye RA: Effects of fentanyl,

droperidol, and Innovar on canine cerebral meta-

bolism and blood flow. Br J Anaesth 43:630-636,

1971.

107. Strebel S, Kaufmann M, Maitre L, et al: Effects of

ketamine on cerebral blood flow velocity in humans.

Influence of pretreatment with midazolam or

esmolol. Anaesthesia 50:223-228, 1995.

108. Cavazzuti M, Porro CA, Biral GP, et al: Ketamine

effects on local cerebral blood flow and metabolism

in the rat. J Cereb Blood Flow Metab 7:806-811,

1987.

109. Vollenweider FX, Leenders KL, Oye I, et al: Diffe-

rential psychopathology and patterns of cerebral

glucose utilisation produced by (

S

)- and (

R

)-keta-

mine in healthy volunteers using positron emission

tomography (PET). Eur Neuropsychopharmacol

7:25-38, 1997.

110. Vollenweider FX, Leenders KL, Scharfetter C, et al:

Metabolic hyperfrontality and psychopathology in

the ketamine model of psychosis using positron

emission tomography (PET) and [

18

F] fluorodeoxy-

glucose (FDG). Eur Neuropsychopharmacol 7:9-24,

1997.

111. Holcomb HH, Lahti AC, Medoff DR, et al: Sequen-

tial regional cerebral blood flow brain scans using

PET with H

2

15

O demonstrate ketamine actions in

CNS dynamically. Neuropsychopharmacology 25:

165-172, 2001.

112. Schmidt A, Ryding E, Akeson J: Racemic ketamine

does not abolish cerebrovascular autoregulation in

the pig. Acta Anesthesiol Scand 47:569-575, 2003.

113. Mayberg TS, Lam AM, Matta BF, et al: Ketamine

does not increase cerebral blood flow velocity or

intracranial pressure during isoflurane/nitrous

oxide anesthesia in patients undergoing craniotomy.

Anesth Analg 81:84-89, 1995.

114. Strebel S, Kaufmann M, Maître L, et al: Effects of

ketamine on cerebral blood flow velocity in humans.

Influence of pretreatment with midazolam or

esmolol. Anaesthesia 50:223-228, 1995.

115. Sakai K, Cho S, Fukusaki M, et al: The effects of

propofol with and without ketamine on human

cerebral blood flow velocity and CO

2

response.

Anesth Analg 90:377-382, 2000.

116. Albanese J, Arnaud S, Rey M, et al: Ketamine

decreases intracranial pressure and electroencepha-

lographic activity in traumatic brain injury patients

during propofol sedation. Anesthesiology 87:1328-

1334, 1997.

117. Sakabe T, Maekawa T, Ishikawa T, et al: The effects

of lidocaine on canine cerebral metabolism and

circulation related to the electroencephalogram.

Anesthesiology 40:433-441, 1974.

118. Astrup J, Sorensen PM, Sorensen HR: Inhibition of

cerebral oxygen and glucose consumption in the

dog by hypothermia, pentobarbital, and lidocaine.

Anesthesiology 55:263-268, 1981.

119. Lam AM, Donlon E, Eng CC, et al: The effect of

lidocaine on cerebral blood flow and metabolism

during normocapnia and hypocapnia in humans

[abstract]. Anesthesiology 79:A202, 1993.

120. Bedford RF, Persing JA, Pobereskin L, et al: Lido-

caine or thiopental for rapid control of intracra-

nial hypertension? Anesth Analg 59:435-437,

1980.

121. Michenfelder JD, Milde JH: Influence of anesthetics

on metabolic, functional and pathological respon-

ses to regional cerebral ischemia. Stroke 6:405-410,

1975.

122. Michenfelder JD, Sundt TM, Fode N, et al: Isoflu-

rane when compared to enflurane and halothane

decreases the frequency of cerebral ischemia during

carotid endarterectomy.Anesthesiology 67:336-340,

1987.

123. Todd MM, Drummond JC: A comparison of the

cerebrovascular and metabolic effects of halothane

and isoflurane in the cat. Anesthesiology 60:276-

282, 1984.

124. Lutz LJ, Milde JH, Milde LN: The cerebral functio-

nal, metabolic, and hemodynamic effects of des-

flurane in dogs. Anesthesiology 73:125-131, 1990.

125. Scheller MS, Tateishi A, Drummond JC, et al: The

effects of sevoflurane on cerebral blood flow, cere-

bral metabolic rate for oxygen,intracranial pressure,

and the electroencephalogram are similar to those

of isoflurane in the rabbit. Anesthesiology 68:548-

551, 1988.

126. Michenfelder JD, Cucchiara RF: Canine cerebral

oxygen consumption during enflurane anesthesia

and its modification during induced seizures.Anes-

thesiology 40:575-580, 1974.

127. Drummond JC, Todd MM, Scheller MS, et al: A

comparison of the direct cerebral vasodilating

potencies of halothane and isoflurane in the New

Zealand white rabbit. Anesthesiology 65:462-467,

1986.

128. Hansen TD, Warner DS, Todd MM, et al: Distribu-

tion of cerebral blood flow during halothane versus

isoflurane anesthesia in rats.Anesthesiology 69:332-

337, 1988.

129. Lenz C, Rebel A, Klaus V, et al: Local cerebral blood

flow, local cerebral glucose utilization, and flow-

metabolism coupling during sevoflurane versus

isoflurane anesthesia in rats. Anesthesiology 89:

1480-1488, 1998.

130. Heath KJ, Gupta S, Matta BF: The effects of sevo-

flurane on cerebral hemodynamics during propofol

anesthesia. Anesth Analg 85:1284-1287, 1997.

131. Wollman H,Alexander SC, Cohen PJ, et al: Cerebral

circulation of man during halothane anesthesia.

Anesthesiology 25:180-184, 1964.

100

Fisiología y anestesia

I